Do children need more vaccines? RFK thinks so...

lemonhelmet · 3/12/2025, 1:05:51 PM · permalink

i was not a skeptic until my son got injured 15 years ago. i was so not a skeptic that they gave him polio, DTaP and MMR on the same day. in the first 4 hours, he developed facial asymmetry, in the next 48h he lost words, and muscle tone and stopped looking at me and recognizing me. at 3 he was running, screaming and breaking things 22h a day and slept only 2. Food was reduced to hot dogs, pizza and soup. i could not get any answers from the medical staff so i took matters into my hands... i had motivation like never in my life, so i took medical books, courses, listened to lectures, mapped out every single symptom he had and targeted those symptoms. now he is 16 communicates in 2 languages functionally, is self-reliant. this is my opinion now:

In a scientific article, I read that human DNA transfers environmental memory for 14 generations. Interestingly, in Serbia, we have a family tree organized for 16 generations, which aligns closely with what I read. The oldest in our lineage are known as White Eagle (male) and White Bee (female), so my son and I would have different White Eagle and White Bee ancestors.

I believe that over the last 200 years, rapid changes in our lifestyle have been detrimental to our health. We no longer eat the same food or store it in the same way. We are exposed to artificial lights and sounds, and our sleep patterns have changed. Additionally, the products we use on our bodies, including fabrics, have drastically evolved.

While I believe the human body can withstand and adapt to some changes over 14 or 16 generations, I don't think our DNA is designed to endure so many changes in just a few generations. Furthermore, the substances we inject into infants add to this burden. Together, these factors contribute to the rise of many "mysterious" conditions.

I have to add to this the case of Hannah Poling. Hannah was born in 2004 and because her parents are in the medical field ( her father is a neurologist John Poling) she was in a study of healthy children up until she got MMR and regressed into autism. Her case was proven in court but sealed because she was a minor ( even though it is a public concern case). That is when they found mitochondrial dysfunction as a predisposition to regressive autism after shots as well as that over 36% of the population born has mitochondrial dysfunction. I believe we are poisoning our bodies for the last 200 years by attacking mitochondria.

P.S. I believe that autism is a cluster of different mechanisms of damage and that is where DNA kicks in and why they all seem to have the same but totally different symptoms. And that children do not have those symptoms because they have autism but rather they have autism because of those symptoms

The Chronological Flow and Branching of Autism Causes

Autism, as a complex neurological disorder, stems from the interplay of genetic, epigenetic, and environmental factors. Its causal chain can be traced from fundamental DNA damage and epigenetic modifications, through metabolic disruptions, to specific mechanisms such as mitochondrial dysfunction, ischemia, blood-brain barrier (BBB) permeability, and, ultimately, neurological imbalances. Each of these points branches out and impacts subsequent processes, making the development of autism cumulative and causally interlinked.

1\. Starting Point: DNA Damage and Epigenetic Mechanisms

Epigenetic processes, such as DNA methylation and histone modification, allow the body to adapt to environmental changes without altering the core genetic structure. However, DNA damage caused by toxins, inflammation, or nutrient deficiencies disrupts this delicate regulation.

Cause: Industrialization and increased exposure to heavy metals, pesticides, and microplastics.

Consequence: Improper DNA methylation hampers the expression of genes crucial for neurological development.

Further Branching: Damaged DNA transitions into mitochondrial dysfunction, heightening the organism’s sensitivity to metabolic stress.

2\. Impaired DNA Methylation and Folate Cycle Disruption

DNA methylation relies on the availability of folate, vitamin B12, and other essential substances. When these nutrient reserves are depleted, or the methylation process is disrupted:

Cause: Poor nutrition, genetic mutations like MTHFR, and chemical imbalances.

Consequence: Gene expression dysfunction impacts the immune system and nervous system development.

Further Branching: Methylation imbalances lead to mitochondrial dysfunction, increased oxidative stress, and metabolic issues.

3\. Mitochondrial Dysfunction: The Core of Metabolic Issues

Mitochondria are vital for energy production and cellular detoxification. When they malfunction:

Cause: Inherited mutations, toxin exposure, and oxidative stress.

Consequence: Energy deficiency impairs neuronal function and causes neuroinflammation.

Further Branching: Mitochondrial dysfunction triggers localized ischemic processes and compromises the blood-brain barrier.

4\. Ischemia and Inflammatory Responses

Ischemia – reduced blood flow to brain regions – increases the risk of neuronal damage. Concurrently, inflammation exacerbates damage to brain cells and structures.

Cause: Mitochondrial dysfunction and impaired circulation.

Consequence: Chronic inflammatory responses compromise the structural integrity of the blood-brain barrier (BBB).

Further Branching: A leaky BBB allows toxins and immune cells to infiltrate the brain.

5\. Blood-Brain Barrier (BBB) Permeability

The BBB acts as a protective layer, preventing harmful substances from entering the brain. When its function is disrupted:

Cause: Inflammation, oxidative stress, and toxic influences.

Consequence: Toxins, microglia, and immune markers enter the brain, exacerbating neuroinflammation.

Further Branching: Toxin and metal ion infiltration initiates calcium accumulation and synaptic dysfunction.

6\. Calcium Accumulation and Synaptic Dysfunction

Excessive calcium buildup in neurons disrupts synaptic function, directly affecting neuronal plasticity and signaling.

Cause: Dysregulation of calcium channels due to oxidative stress and ischemia.

Consequence: Synaptic dysfunction hinders communication between brain regions, contributing to autistic symptoms.

Further Branching: Prolonged synaptic dysfunction leads to lasting neurological damage.

7\. Final Outcome: Neurological and Cognitive Symptoms

All previous branches, from DNA damage to synaptic dysfunction, culminate in the manifestation of autistic symptoms:

Challenges in social communication.

Stereotyped behaviors and sensory hypersensitivity.

Cognitive imbalances and reduced flexibility in thinking.

These manifestations are not the result of a single cause but a complex web of interconnected biological and environmental factors.

Conclusion

The development of autism can be understood as the result of a cause-and-effect chain, beginning with DNA damage and epigenetic processes, through methylation and mitochondrial dysfunction, and culminating in neurological symptoms. Each point in this chain branches into intricate mechanisms, each contributing to the final picture. Understanding these interdependencies paves the way for precise diagnostics, prevention, and treatment of autism, with a particular emphasis on an individualized approach to care.

We have exchanged natural cause for dna damages.... we saved a lot but destroyed humanity

5 bumps